Eczema Herpeticum in Atopic Dermatitis: Signs, Stages, and Urgent Care

Eczema Herpeticum in Atopic Dermatitis: Signs, Stages, and Urgent Care
By Michael Anderson, Clinical Research Project Manager •

You know your own eczema. You know how it itches, where it lands, and how long it takes to settle down. So when a patch erupts overnight into a tight cluster of tiny blisters that all look the same size, pay attention. That pattern is the signature of eczema herpeticum, a herpes simplex virus infection spreading across skin that atopic dermatitis already damaged.[1]

Here is what makes it feel wrong. It burns instead of itching, a fever shows up by morning, and your usual cream does nothing. Those whole-body signs are common in this infection, which is why it does not behave like the flares you have handled for years.[2]

This guide walks you through how to tell this infection apart from your ordinary flare, what the stages look like day by day, what treatment actually involves, and how to lower your odds of a repeat. It is a complication, not one of atopic dermatitis and the other recognized types of eczema.

One point drives everything below. Antiviral treatment started early changes the course of the illness, and in children hospitalized with it, each day of delay in starting acyclovir was linked with a longer hospital stay.[3]

Key Takeaways

  • Eczema herpeticum is a herpes simplex virus infection spreading across eczema-damaged skin.
  • It is not a type of eczema. It is a complication needing urgent antiviral treatment.
  • Clustered same-sized blisters, pain instead of itch, and fever are the warning signs.
  • Severe, early-onset, head-and-neck eczema carries the highest risk.
  • Controlling baseline inflammation and barrier damage lowers your risk of another episode.

Table of Contents

What Is Eczema Herpeticum?

⚠️ This is a same-day medical problem:

If you suspect eczema herpeticum, get seen today rather than waiting to watch it. Antiviral treatment works best when it starts early.[3]

Eczema herpeticum is a widespread herpes simplex virus infection of skin already damaged by eczema. The virus slips in through breaks in the barrier and spreads across large areas, raising clusters of small identical blisters that sting under a shirt collar, along with pain and often fever. Doctors also call it Kaposi varicelliform eruption.[1]

The word herpeticum points straight at the culprit, herpes simplex virus. Most cases involve HSV-1, the same virus behind the cold sore that tingles on a lip a day before it blisters, while HSV-2 causes it far less often.[4]

One question comes up constantly: is atopic dermatitis the same thing as eczema? In everyday use, yes, because atopic dermatitis is the most common form of eczema and the two names describe one condition, as our guide to atopic eczema and atopic dermatitis explains.

Eczema herpeticum is not a type of eczema. It is a complication of one. Other conditions that break the skin barrier can also set the stage, including burns, severe seborrheic dermatitis, and rarer blistering or inherited skin diseases such as pemphigus and Darier disease. Atopic dermatitis, though, is by far the most commonly reported underlying condition.[5] It stays uncommon, yet it can become severe and even life-threatening when it goes untreated.[6]

So why does one virus behave so differently on eczema skin? The answer sits in three layers of your skin's defense, and knowing which layer is failing tells you which parts of your risk you can actually change.

Why Atopic Dermatitis Skin Is Vulnerable to the Herpes Virus

Healthy skin usually stops herpes simplex virus at a small landing site. A cold sore stays a cold sore. Eczema skin loses that containment on three separate fronts at once, which is why the same virus can cover a shoulder, a neck, or a whole face.

Cross-section comparing healthy skin with atopic dermatitis skin showing three vulnerability layers that let herpes simplex virus cause eczema herpeticum

A broken barrier gives the virus a wide-open door

Your outer skin layer works like mortar between bricks. In atopic dermatitis that mortar is thin, cracked, and leaky, which gives the virus far more surface area to work with than it would find on healthy skin.

Scratching widens the opening. Fingers carry virus from one raw patch to another in a process called autoinoculation, which helps explain the scattered, spreading pattern.[8] The same broken surface also favors heavy Staphylococcus aureus colonization, so bacterial and viral infection often travel together.[9] Our guide to antibiotics for infected eczema covers that bacterial side.

Why the eczema immune response fails to contain HSV

Your immune system has different settings, and atopic skin is stuck on the wrong one. It runs what doctors call a Th2-dominant program, the branch of immunity built to fight parasites and drive allergy, steered by two signaling proteins called IL-4 and IL-13. That setting handles viruses poorly, so herpes simplex arrives to find a guard trained for the wrong kind of intruder.

Those same signals also mute your antimicrobial peptides, the natural antibiotics your skin makes for itself. Two of them, cathelicidin LL-37 and the human beta-defensins, normally break viruses apart within hours of contact, much like sprinklers dousing a fire before the trucks arrive. In atopic dermatitis inflamed skin produces far less of them, and that shortfall is most pronounced in people who have already had eczema herpeticum, so an early line of defense is simply missing.[7]

Put those two problems together and the virus gets a head start it never gets on healthy skin, which is why a sore that would stay the size of a pea on someone else can sweep across a whole cheek here.

Who is most at risk of eczema herpeticum

Risk factors that show up repeatedly in eczema herpeticum research:

  • Severe, widespread eczema: higher severity scores and greater affected body surface area track with higher risk.[10]
  • Early age of onset: eczema that started in the first year of life shows up disproportionately in affected groups.[10]
  • High total IgE: high levels of IgE, the antibody behind allergic reactions, turn up repeatedly in this research.[10]
  • Head and neck involvement: eczema concentrated on the face and neck sits closest to where HSV lives.[10]
  • A previous episode: recurrence is uncommon but does happen, so a prior episode is worth flagging to your clinician.[10]
Radial chart of eczema herpeticum risk factors in atopic dermatitis including severity, early onset, high IgE, and head and neck involvement

What this means for your skin: severity is the risk factor you can actually move, because the more skin you leave inflamed and broken, the more landing surface the virus gets.

Knowing you sit in the higher-risk group only pays off if you can recognize the eruption quickly, because antivirals work best when they start early. That window opens in the first day or two, and here is what it looks like.

Early Signs and Stages of Eczema Herpeticum

Picture standing at the mirror at 6 a.m. and counting twice as many blisters as you saw at midnight, with your collar already sticking to the sore spots. That speed is a clue in itself, because a normal flare does not double overnight.

The first 24 to 48 hours: early signs of eczema herpeticum

If you do only one thing: photograph the area and get seen today rather than waiting to see whether it spreads.

  1. Uniform blisters: clusters of small blisters on or beside eczema, all roughly the same size and shape.[11]
  2. Pain, not itch: burning or stinging that feels out of proportion to your usual itching.[11]
  3. Fast spread: new crops appearing over hours to a day or two, not weeks.[11]
  4. Feeling unwell: fever, chills, tiredness, and swollen lymph nodes near the area.[2]
  5. A flare that stops responding: your usual routine suddenly does nothing at all.[14]
  6. Punched-out sores: blisters collapsing into round, sharply edged erosions, as if a paper punch had lifted a disc of skin.[11]

Compare what you see against your own baseline. Our guide to typical eczema symptoms is a useful reference point.

Eczema herpeticum stages, from first blister to healing

Once it starts, the infection follows a fairly predictable arc. Blisters appear, break down, crust over, and heal, usually across two to three weeks with antiviral treatment, though widespread cases can take longer.[12]

Before visible symptoms

Incubation. Nothing visible yet, though the virus is already establishing itself.

Days 1 to 2

Crops of small, same-sized, fluid-filled blisters appear on eczematous skin.[11]

Days 3 to 7

Blisters turn cloudy with pus and sink in at the center. Fever and that flattened, achy feeling usually peak here.[2]

Week 2

Roofs shear off, leaving round punched-out erosions that merge and crust.[11]

Weeks 2 to 3

Crusts separate and skin heals; scarring is a recognized longer-term outcome in some cases.[10]

Eczema herpeticum stages timeline from herpes virus exposure through blistering, crusting, and healing over two to three weeks

That tidy timeline assumes treatment. Left alone, extensive cases tend to keep progressing instead of resolving, with a real risk of the virus reaching the bloodstream.[6]

Where it appears, and why babies need faster care

The face, head, neck, and upper trunk are the classic sites, which mirrors where HSV usually lives and where atopic eczema often concentrates.[10] Any actively inflamed skin can be involved, though. If your eczema sits mainly above the collarbone, our facial eczema treatment guide is worth reading alongside this one.

Infants and young children deserve a lower threshold for concern. In one large group of hospitalized children the typical patient was just 1 year old, about 1 in 25 needed intensive care, and roughly a third also had a staph infection on top of the virus.[3] Parents managing baby eczema should treat any sudden crop of uniform blisters as urgent.

⚠️ Blisters near the eye are an emergency:

Herpes can infect the cornea, the clear window at the front of the eye, and threaten vision, so eye pain, light sensitivity, or blurred vision needs same-day care from an eye specialist.[13]

Knowing the pattern is only half the job, because several far less urgent skin problems can look similar at first glance. Telling them apart is what decides whether you need an antiviral tonight or just your usual cream.

Eczema Herpeticum vs. Eczema Flare and Other Look-Alikes

The real question, standing at the bathroom mirror at 11 p.m., is whether this is the same trouble you have had before or something new. Misreading this infection as a bad flare or a simple bacterial infection is common, and that mistake delays antivirals.[14] The table below lines up the conditions most often confused with each other.

Condition What the lesions look like Fever? How fast it spreads What it needs
Eczema herpeticum Clusters of same-sized blisters, then round punched-out sores[11] Often Hours to days Urgent systemic antivirals
Typical eczema flare Ill-defined dry itchy patches, scaling, thickened skin No Days to weeks Usual anti-inflammatory and moisturizer
Weeping eczema Raw oozing areas with clear fluid, no discrete uniform blisters Usually no Days Barrier care, sometimes antibacterial
Impetigo or staph infection Pustules with honey-colored golden crust[15] Sometimes Days Antibacterial treatment
Dyshidrotic eczema Small, deep-seated vesicles on the palms, finger sides, or soles No Days Topical anti-inflammatory
Scabies Burrows and papules in finger webs and waistline, night itch[16] No Weeks, spreads in households Scabicide for everyone at home
Molluscum contagiosum Firm dome-shaped papules with a central dimple[17] No Weeks to months Often watchful waiting
Comparison chart of eczema herpeticum versus a typical eczema flare, weeping eczema, and dyshidrotic blisters

Two rows deserve a note. Weeping skin oozes clear serum from broad raw areas, which is different from discrete same-sized domes, and our article on drying out eczema explains why that distinction matters. When an itchy, bumpy eruption appears, the question of scabies or eczema also comes up often, and burrows plus household spread usually settle it.[16]

One more mimic is follicular eczema, a form of atopic dermatitis that raises small bumps around individual hair follicles. It can pass for early herpes blisters because both show up as crops of matching bumps.[18]

📚 Related Resource

See our guide: What is Dyshidrotic Eczema? Complete Treatment Guide

What a biopsy or bedside test shows

Sometimes a pathology report settles the argument. Classic atopic dermatitis eczema histology shows spongiosis, meaning fluid pushing skin cells apart, along with inflammatory cells in the upper skin.[19]

Herpes infection looks entirely different under the microscope. Pathologists see the damage the virus does to individual cells: skin cells swollen like overfilled water balloons, cells fused together with several nuclei apiece, and glassy specks inside those nuclei. A Tzanck smear, a quick scraping from a fresh blister that is stained and read right in the office, can reveal those same fused cells within minutes.[20]

In practice, though, most people never need a biopsy, because the diagnosis is usually made from the pattern on the skin itself. Knowing how that appointment unfolds takes some of the dread out of going in.

How Eczema Herpeticum Is Diagnosed and Treated

You will probably not wait days for a lab result. Most clinicians diagnose this by looking, because known eczema plus a sudden crop of same-sized blisters plus a fever is a pattern distinctive enough to start treatment while the swabs are still at the lab.[1] Expect the visit to move fast once someone recognizes it.

Tests that confirm eczema herpeticum

  • Viral PCR swab: a swab rubbed on the base of a fresh blister and tested for the virus's genetic material, which is the usual way the diagnosis is confirmed.
  • Tzanck smear: fast and cheap at the bedside, though it misses some cases and cannot tell herpes apart from varicella zoster, the chickenpox and shingles virus.[20]
  • Direct fluorescent antibody and viral culture: useful backups that tag or grow the virus from a sample, though culture takes longer and can miss older, crusted sores.
  • Bacterial swab: usually taken at the same visit, since a bacterial infection layered on top of the viral one is common.[6]
  • Skin biopsy: reserved for unclear cases where the picture does not fit.[20]

Antiviral treatment: what to expect

What actually clears this is an antiviral taken by mouth or given through a drip, not one rubbed on the skin. Acyclovir is the established first-line drug, with valacyclovir as an oral alternative, and a cream version will not do the job here because the medicine has to travel through your bloodstream to reach every infected patch.[21] Treating this with an ointment would be like watering one corner of a garden that is drying out everywhere.

If you are otherwise well, oral therapy at home is often enough. Extensive disease, high fever, vomiting, infancy, or a weakened immune system usually push treatment toward an intravenous route in hospital.[22] Antibiotics get added when a bacterial infection is layered on top, and any eye involvement brings an eye specialist in urgently.[6][13]

Supportive care matters too: pain relief, fluids, and gentle wound care while the skin repairs. Dosing is a decision for your prescriber, not something to guess at from an article.

Signs that usually mean hospital care:

  • Widespread involvement: large body surface area or rapid extension beyond eczematous skin.[22]
  • Systemic illness: high fever, dehydration, or an inability to keep oral medicine down.[22]
  • Very young age or immunosuppression: both groups face a higher complication rate.[3]

Should you keep using your eczema cream during an outbreak?

Ask your treating clinician, and ask before you apply anything. Antiviral therapy is the priority and nothing should delay it. There is no clear published guidance on continuing topical anti-inflammatories during an active infection, so this is a supervised decision rather than a self-directed one.

One rule holds regardless: do not put topical products on open, broken, or bleeding skin, a principle covered in our help article on where SmartLotion is safe to apply. Wait until the surface has closed over again.

When to seek emergency care

Go now, not tomorrow, if any of the following applies, because each one suggests the infection may be moving beyond the skin:

  • Eye symptoms: blisters near the eye, eye pain, light sensitivity, or changed vision.[13]
  • High or rising fever: especially with confusion, shaking chills, or severe malaise.[6]
  • Rapid spread: new blisters appearing on skin that never had eczema.[11]
  • An unwell infant: lethargy, poor feeding, or fewer wet diapers.[3]
  • No improvement: continued worsening after antiviral treatment has started.[3]
Eczema herpeticum emergency warning signs infographic listing six red flags that require urgent medical care

Getting through the acute illness is the hard part, and once your skin has closed over the useful question becomes how to keep the virus from finding that same opening again.

Lowering Your Risk of Another Episode

Once you have recovered, the obvious question is how to avoid a repeat. Recurrence is uncommon but possible, so it is worth planning for rather than hoping about.[10] Two tracks matter: less virus reaching you, and less vulnerable skin waiting for it.

Reducing your exposure to the herpes virus

If you do only one thing: keep away from active cold sores, including a well-meaning kiss on a child's cheek.

  • Avoid direct contact: herpes simplex spreads through skin-to-skin contact, so keep away from active cold sores and other visible lesions.
  • Do not share: lip balms, towels, razors, and drinking glasses all move virus between people.
  • Wash hands first: clean hands before touching or treating eczematous skin reduce autoinoculation.[8]
  • Keep nails short: less scratching damage means fewer entry points.[8]
  • Ask about suppressive therapy: daily suppressive antivirals reduce recurrences in people with frequent HSV outbreaks, though that trial evidence comes from recurrent herpes labialis rather than eczema herpeticum specifically.[23]

One extra caution belongs here. People with active atopic dermatitis and their close household contacts are advised against live smallpox vaccination because of the risk of eczema vaccinatum, a related and serious complication.[25]

Getting your baseline eczema under better control

Here is the part you can actually control. Because risk tracks with how severe and how widespread your eczema is, calmer skin is genuinely better protected skin. Regular use of emollients, the rich moisturizers that seal water into the skin, lowers eczema severity and cuts how often flares arrive, which is why moisturizing twice daily counts as non-negotiable foundational care at every severity level, during flares and between them.[24] Treating inflammation steadily rather than chasing flares after they start also reduces relapses.[26] Think of it as repointing the mortar between the bricks a little at a time, instead of waiting for the wall to crumble.

Over-the-counter options fall into four separate groups, and they are not interchangeable:

  • Plain moisturizers: restore water and lipids, no anti-inflammatory action.
  • Prebiotic moisturizers: support the skin microbiome alongside hydration.
  • OTC 1% hydrocortisone: a mild option some people use for limited periods on mild eczema; check with a clinician about how long to use it.
  • SmartLotion: its own category, combining anti-inflammatory action, prebiotic microbiome support, and moisturization in one formulation.

That last category is why SmartLotion, developed by a board-certified dermatologist at HarlanMD, is formulated as an eczema cream intended for daily control across severities, ages, and sensitive areas including the face. To be explicit: it does not treat a viral infection, and no topical belongs on open or broken skin. Wait until your skin has healed.

Once you are back to intact skin, the goal shifts to staying there. Because it is designed for long-term daily use, SmartLotion functions as an eczema treatment cream you keep using between flares, not only during them. Our help article on the adult atopic dermatitis body protocol lays out that maintenance routine.

📚 Related Resource

See our guide: Atopic Dermatitis Treatments

Better baseline control will not make you immune, but it shrinks the surface the virus can use.

Frequently Asked Questions About Eczema Herpeticum

How did I get eczema herpeticum?

You picked up herpes simplex virus, usually through close contact with someone carrying a cold sore, which then spread across eczema-damaged skin.

Is eczema herpeticum contagious?

The virus involved is contagious through direct skin contact, so avoid touching the affected area and sharing towels or bedding until it heals. For a fuller answer on transmission and eczema, see our article on whether eczema can spread.

Is eczema herpeticum an STD?

No. Most cases come from HSV-1, the ordinary cold sore virus that spreads through everyday contact such as kisses and shared cups. HSV-2 can be involved in a minority of cases, but eczema herpeticum itself is not a sexually transmitted infection.[4]

Does eczema herpeticum go away on its own?

Do not wait to find out. Treated promptly, it typically clears over roughly two to three weeks, though some cases take longer.[12] Untreated, it can spread widely, reach the bloodstream, and cause serious complications, which is why prompt antiviral treatment is the standard approach.[6]

Why is eczema herpeticum dangerous?

Large areas of skin lose their protective function at once. That opens the door to bacterial superinfection, fluid loss, and virus entering the bloodstream.[6] Eye involvement can threaten sight, and infants become systemically unwell faster than adults do.[13]

Can eczema herpeticum come back?

Yes, though recurrence is uncommon. Herpes simplex stays in the body permanently, so repeat episodes do happen, particularly when eczema stays severe.[10] If you have had more than one episode, ask your dermatologist about long-term suppressive antiviral therapy.[23]

References

  1. Garg G, Thami GP. "Psoriasis Herpeticum due to Varicella Zoster Virus: A Kaposi's Varicelliform Eruption in Erythrodermic Psoriasis." Indian Journal of Dermatology. 2012 May-Jun;57(3):213–214. View Study
  2. Pedro M, Caldas M, Neves F, Diogo S, Fortunato F. "Eczema Herpeticum Complicating Atopic Dermatitis: A Rare Presentation." Cureus. 2024 Feb 28;16(2):e55171. View Study
  3. Aronson PL, Yan AC, Mittal MK, Mohamad Z, Shah SS. "Delayed Acyclovir and Outcomes of Children Hospitalized With Eczema Herpeticum." Pediatrics. 2011 Dec;128(6):1161–1167. View Study
  4. Chin AG, Asif M, Hultman C, Caffrey J. "Hailey-Hailey Disease with Superimposed Eczema Herpeticum Caused by Herpes Simplex Virus Type 2 Infection in a Burn Unit: A Case Report and Literature Review." Cureus. 2019. View Study
  5. Huang Y-W, Chen C-B, Ger T-Y. "Kaposi varicelliform eruption in Darier disease." Dermatologica Sinica. 2025;43(2):140-141. View Study
  6. Karpierz K, Kuchar EP. "An Infant With Seborrhoeic Dermatitis and Eczema Herpeticum Complicated by a Generalized Infection." Cureus. 2021. View Study
  7. Hata TR, Kotol P, Boguniewicz M, et al. "History of eczema herpeticum is associated with the inability to induce human β-defensin (HBD)-2, HBD-3 and cathelicidin in the skin of patients with atopic dermatitis." The British Journal of Dermatology. 2010 Jun;163(3):659–661. View Study
  8. Peterson AR, Nash E, Anderson BJ. "Infectious Disease in Contact Sports." Sports Health. 2018 Aug 14;11(1):47–58. View Study
  9. Broccardo CJ, Mahaffey S, Schwarz J, et al. "Comparative Proteomic Profiling of Atopic Dermatitis Patients Based on History of Eczema Herpeticum Infection and Staphylococcus aureus Colonization." Journal of Allergy and Clinical Immunology. 2011 Jan;127(1):186-194. View Study
  10. Yazici ED, Foster ME. "Eczema Herpeticum." Don't Forget the Bubbles. 2026. View Study
  11. Satasia M. "Papulovesicular Riddle in an Atopic Individual." Cureus. 2024. View Study
  12. Sharma N, Shrivastava H, Joshi S, Kushwaha RK, Jain SK. "Clinical Insights of Kaposi's Varicelliform Eruption Associated with Underlying Primary Dermatoses- A Case Series from Hadoti Region of Rajasthan." Indian Journal of Dermatology. 2025;70(5):287–291. View Study
  13. Rowe AM, St Leger AJ, Jeon S, Dhaliwal DK, Knickelbein JE, Hendricks RL. "Herpes Keratitis." Progress in Retinal and Eye Research. 2013 Jan. View Study
  14. Lanka P, Woloski JR. "Eczema Herpeticum Misdiagnosed as Facial Cellulitis in an African American Male." Cureus. 2024. View Study
  15. Bowen AC, Tong SYC, Chatfield MD, Carapetis JR. "The microbiology of impetigo in Indigenous children: associations between Streptococcus pyogenes, Staphylococcus aureus, scabies, and nasal carriage." BMC Infectious Diseases. 2014. View Study
  16. Veraldi S, Schianchi R, Nazzaro G. "Scabies and nocturnal pruritus: preliminary observations in a group of African migrants." The Journal of Infection in Developing Countries. 2021. View Study
  17. Atique A, Kanou M, Leclerc N, et al. "Eczematous molluscum contagiosum masquerading as herpes simplex in an 8-year-old: A case report." SAGE Open Medical Case Reports. 2026. View Study
  18. Gutte RM. "Adult Onset Atopic Dermatitis with Secondary Follicular Mucinosis with Cyclosporine Induced Spiny Follicular Hyperkeratosis and Hair Casts." Indian Journal of Dermatology. 2013. View Study
  19. Tanei R, Hasegawa Y. "Immunological Pathomechanisms of Spongiotic Dermatitis in Skin Lesions of Atopic Dermatitis." International Journal of Molecular Sciences. 2022. View Study
  20. Athmanathan S, Pranesh VM, Pasricha G, Garg P, Vemuganti GK, Sharma S. "Atypical Herpes simplex keratitis (HSK) presenting as a perforated corneal ulcer with a large infiltrate in a contact lens wearer: multinucleated giant cells in the Giemsa smear offered a clue to the diagnosis." BMC Ophthalmology. 2001. View Study
  21. Martínez-Ortega JI, Franco González S. "Eczema Herpeticum: Clinical Insights and Pathogenesis Hypotheses on Basolateral Adhesion Proteins." Cureus. 2024. View Study
  22. Naumeri F, Rijal S, Rashid AR, Ahmad HM. "Eczema Herpeticum in children with burns." Pakistan Journal of Medical Sciences. 2021 Mar-Apr;37(2):421–425. View Study
  23. Mancini A, Inchingolo AM, Marinelli G, et al. "Topical and Systemic Therapeutic Approaches in the Treatment of Oral Herpes Simplex Virus Infection: A Systematic Review." International Journal of Molecular Sciences. 2025;26(17):8490. View Study
  24. van Zuuren EJ, Fedorowicz Z, Christensen R, Lavrijsen APM, Arents BWM. "Emollients and moisturisers for eczema." Cochrane Database of Systematic Reviews. 2017 Feb 6;2017(2):CD012119. View Study
  25. Knitlova J, Hajkova V, Voska L, et al. "Development of eczema vaccinatum in atopic mouse models and efficacy of MVA vaccination against lethal poxviral infection." PLoS One. 2014;9(12):e114374. View Study
  26. Kasai H, Kawasaki H, Fukushima-Nomura A, et al. "Stratification of atopic dermatitis patients by patterns of response to proactive therapy with topical tacrolimus: low serum IgE levels and inadequately controlled disease activity at the start of treatment predict its failure." Annals of Medicine. 2021;53(1):2207–2216. View Study

About the Author: Michael Anderson, Clinical Research Project Manager

Michael bridges the gap between research labs and real patients. As our research project manager, he ensures groundbreaking studies translate into accessible treatments. A craft beer enthusiast and woodworking hobbyist, Michael approaches both his hobbies and research with the same attention to detail, although he admits that research protocols are significantly less forgiving than furniture joints.