Eczema Scalp Treatment: Evidence-Based Solutions That Work

Eczema Scalp Treatment: Evidence-Based Solutions That Work
By Jessica Arenas, Lead Research Analyst •

Jennifer tried 14 different shampoos before finding relief from her scalp eczema. She represents approximately 5.6% of adults who experience seborrheic dermatitis, the most common form of scalp eczema[1]. Like many eczema patients who develop symptoms on their scalp[2], she thought nothing would work. Then she discovered the overlooked connection between scalp microbiome and inflammation. Here's what changed everything.

The journey through scalp eczema treatments often feels endless. You've likely tried medicated shampoos, topical steroids, natural oils, and maybe even prescription antifungals. Each new product brings hope, then disappointment when the itching and flaking return. Research confirms that treatment adherence remains a significant challenge in dermatological conditions, with many patients struggling to maintain consistent treatment regimens[3]. The constant visibility of scalp symptoms adds another layer of frustration, affecting quality of life in many cases[4]. You know your triggers, you've memorized ingredient lists, yet the cycle continues.

This guide reveals evidence-based strategies for breaking the scalp eczema cycle. You'll discover why traditional treatments often fail many chronic sufferers[5]. We'll explore how the scalp's unique microbiome influences treatment success, and why addressing both inflammation and microbial balance is essential for long-term control. You'll learn the crucial differences between scalp eczema types and why this matters for your treatment. Plus, we'll examine proven approaches for seborrheic dermatitis that address root causes rather than just symptoms. Every recommendation comes from peer-reviewed research, not marketing claims.

Recent systematic reviews highlight that combination therapy targeting both inflammation and yeast overgrowth shows promise in treatment-resistant scalp eczema cases[6]. This dual-action approach offers real hope, even if you've struggled with conventional treatments for years.

Key Takeaways

  • Three types affect the scalp - Seborrheic, atopic, and contact dermatitis each require different treatment approaches
  • Microbiome matters - Malassezia yeast overgrowth is strongly associated with seborrheic dermatitis and dandruff[7]
  • Combination therapy works best - Addressing both inflammation and fungal overgrowth shows promise for treatment-resistant cases[8]
  • Hair loss is reversible - Most eczema-related hair loss recovers within 3-6 months of effective treatment[9]
  • Natural remedies have evidence - Tea tree oil demonstrates antifungal activity against Malassezia in laboratory studies[10]

Table of Contents

Understanding Scalp Eczema: Types and Causes

Scalp eczema isn't a single condition, but rather an umbrella term covering three distinct types of dermatitis. Each type has unique triggers and requires specific treatment approaches. Understanding which type affects you is crucial for effective management.

Seborrheic Dermatitis: The Most Common Culprit

Seborrheic dermatitis affects a significant portion of adults, with higher prevalence than in neonates and children[1]. This condition results from an overproduction of sebum combined with an overgrowth of Malassezia yeast, which naturally exists on everyone's skin[11]. Research shows that people with seborrheic dermatitis have higher levels of this yeast, triggering an inflammatory response that causes the characteristic greasy, yellowish scales[11].

Research Update: Molecular studies have identified that Malassezia species, including M. restricta and M. globosa, are commonly associated with seborrheic dermatitis and dandruff-afflicted scalps, supporting the importance of antifungal treatment[7].

The symptoms extend beyond simple dandruff. Patients experience:

  • Greasy, yellow or white scales that stick to the hair shaft
  • Red, inflamed patches on light skin or lighter patches on darker skin
  • Persistent itching that worsens with stress

Atopic Dermatitis on the Scalp

While atopic dermatitis typically affects skin folds, many patients also develop scalp involvement[2]. This type stems from genetic factors affecting the skin barrier, specifically loss-of-function variants in the filaggrin gene that are strong predisposing factors for atopic dermatitis[12]. The compromised barrier allows increased water loss and allergen penetration, creating a cycle of inflammation.

Unlike seborrheic dermatitis, atopic scalp eczema presents as:

  • Intensely itchy, dry patches rather than greasy scales
  • Clear borders between affected and healthy skin
  • Symptoms that fluctuate with environmental triggers

Contact Dermatitis: The Overlooked Type

Contact dermatitis on the scalp is a common site of involvement in patients with cosmetic contact dermatitis, particularly among those who regularly use hair products[13]. It develops through two mechanisms:

Allergic contact dermatitis occurs after repeated exposure to specific chemicals. Common culprits include paraphenylenediamine (PPD) in hair dyes, which is frequently identified as an allergen in patch testing studies[13], and preservatives like methylisothiazolinone found in many shampoos[14].

Irritant contact dermatitis results from immediate skin damage. Sodium lauryl sulfate (SLS), commonly found in many conventional shampoos, strips natural oils and disrupts the skin barrier[15].

📚 Related Resource

For specific guidance on identifying your triggers, see our guide: Eczema Triggers: Hidden Causes That Make Your Skin Worse

Medical Treatments That Actually Work

Effective scalp eczema treatment requires a strategic approach based on your specific type and severity. Recent clinical trials have identified clear treatment hierarchies with impressive success rates when properly implemented.

Antifungal Shampoos: First-Line Defense

For seborrheic dermatitis, antifungal shampoos remain the gold standard. A systematic review found ketoconazole 2% shampoo has been shown to be beneficial compared to placebo for treating seborrheic dermatitis of the scalp[8]. The treatment works by reducing Malassezia yeast counts and has mild anti-inflammatory properties.

Maintenance therapy with once-weekly ketoconazole can help maintain remission in seborrheic dermatitis[8]

Alternative antifungal options with proven efficacy include:

  • Ciclopirox: Has been shown to be beneficial compared to placebo for treating seborrheic dermatitis of the scalp[8]
  • Selenium sulfide: May be more effective than vehicle shampoo at reducing dandruff severity[8]
  • Zinc pyrithione: May be more effective than vehicle shampoo at reducing dandruff severity, though evidence is limited[8]

Application technique matters. Leaving shampoo on the scalp for several minutes before rinsing can improve effectiveness. For thick scales, pre-treatment with salicylic acid preparations may help improve penetration.

Topical Steroids: Breaking the Inflammation Cycle

When inflammation is severe, topical corticosteroids provide rapid relief. There is consensus that topical corticosteroids are effective in treating seborrheic dermatitis of the scalp[8]. However, treatment duration matters critically.

Treatment duration guidelines recommend limiting continuous use of topical corticosteroids to avoid adverse effects[8]:

  • Mild steroids (1% hydrocortisone): Safe for children and facial extension, use up to 4 weeks
  • Moderate steroids (betamethasone valerate): Effective for scalp, limit to 2 weeks continuous use
  • Potent steroids (clobetasol): Reserve for severe flares, maximum 1 week

Long-term steroid use carries risks. Topical corticosteroid adverse effects fall into three main categories:

  1. Local/Cutaneous Effects: Chronic use of topical corticosteroids is associated with adverse effects, the major one being skin atrophy, which can involve epidermal thinning, decreased keratinocyte number and size, and diminished stratum corneum[16]. Potent steroids like clobetasol propionate can induce measurable skin atrophy after as little as two weeks of treatment under occlusion[16]. Other common local effects include striae, telangiectasias, rosacea, purpura, and acneiform eruptions.
  2. Tachyphylaxis: Reduced effectiveness can develop with continuous use, as keratinocytes develop resistance to glucocorticoid-induced anti-proliferative effects after approximately two weeks of treatment[17].
  3. Systemic Effects: Systemic adverse effects such as adrenal suppression, decreased growth rate, hypertension, hyperglycemia, and Cushing syndrome are rare with low-potency corticosteroids and have low risk in adults if doses do not exceed 50 g per week, even with super-high-potency formulations[18].

Gentle Alternative: Some newer formulations combine low-dose hydrocortisone with prebiotics to address both inflammation and skin microbiome health. Low-potency corticosteroids like hydrocortisone are generally considered safe for extended use when used appropriately[18].

👨‍⚕️ Dr. Harlan's Protocol

For detailed instructions on managing seborrheic dermatitis with SmartLotion, view the official application guide.

Systemic Medications: When Topicals Fail

Severe, widespread scalp eczema may require systemic treatment when topical therapies are insufficient[19]. The BEACON trial, currently underway, compares effectiveness of leading systemic options[20]:

Medication Effectiveness Time to Response Key Considerations
Dupilumab Demonstrates high efficacy in clinical trials and real-world studies[5] 4-6 weeks Injectable, expensive
Methotrexate Effective for severe atopic eczema[19] 8-12 weeks Regular monitoring required
Cyclosporine Effective for severe atopic eczema[19] 2-4 weeks Short-term use only

Evidence-Based Natural Remedies

While conventional treatments form the cornerstone of scalp eczema management, several natural remedies show promising clinical evidence. These work best as complementary therapies rather than replacements for medical treatment.

Tea Tree Oil: More Than Anecdotal

Tea tree oil demonstrates legitimate antifungal properties against Malassezia species in vitro[10]. The oil's effectiveness comes from terpinen-4-ol, which disrupts fungal cell membranes[21]. While clinical trial evidence for scalp application is limited, laboratory studies support its antimicrobial activity against the yeasts associated with seborrheic dermatitis.

Proper dilution is critical to avoid skin irritation. General recommendations include:

  • Mix 1 drop tea tree oil with 12 drops carrier oil for spot treatment
  • Add 2-3 drops to regular shampoo for maintenance
  • Perform patch testing first, as contact allergies can occur[21]

Coconut Oil: Barrier Repair Properties

Virgin coconut oil contains lauric acid with documented antimicrobial effects. Studies have shown coconut oil can reduce Staphylococcus aureus colonization in atopic dermatitis patients[22]. Additionally, coconut oil improves skin hydration when applied twice daily[23].

Application method for scalp treatment:

  1. Warm 1-2 tablespoons until liquid
  2. Part hair in sections and massage into scalp
  3. Cover with shower cap for 30 minutes minimum
  4. Wash with gentle shampoo (may require two washes)

Apple Cider Vinegar: pH Balance

The scalp's normal pH is typically below 5, but eczema may shift this toward alkalinity[24]. Apple cider vinegar, diluted 1:1 with water, may help restore proper pH. While clinical evidence remains limited, some patients report symptom improvement with regular use[25].

Scalp Eczema vs Psoriasis: Critical Differences

Distinguishing between scalp eczema and psoriasis proves challenging, yet crucial for proper treatment. Both conditions affect similar demographics and locations, but key differences guide diagnosis and therapy.

Visual and Symptomatic Distinctions

Psoriasis typically presents as thick, silvery scales with clearly defined borders that often extend beyond the hairline[26]. The scales appear dry and powdery, contrasting with eczema's greasy, yellowish appearance. Histological studies reveal psoriasis plaques are thicker than eczema lesions[27].

Key Difference

Eczema typically causes intense itching, while psoriasis often produces milder itch with burning sensation

Age of Onset Patterns

Seborrheic dermatitis shows bimodal distribution: first peak in infancy and second peak between ages 30-60[28]. Conversely, scalp psoriasis typically manifests between ages 15-35 and is less common in children[29].

Treatment Response Differences

The conditions respond differently to treatments:

  • Antifungals: Effective for seborrheic dermatitis but minimal impact on psoriasis[8]
  • UV Light: Can benefit psoriasis patients but may worsen eczema[19]
  • Vitamin D analogs: First-line for psoriasis, limited benefit for eczema[30]

Addressing Hair Loss and Regrowth

Hair loss from scalp eczema creates significant distress, particularly in patients with moderate to severe cases[31]. Understanding the mechanisms helps set realistic expectations for recovery.

Why Eczema Causes Hair Loss

Research identifies three primary mechanisms:

  1. Inflammation disrupts follicle function: Cytokines released during flares shift follicles into telogen (resting) phase prematurely[32]
  2. Scratching causes mechanical damage: Repeated trauma can lead to follicle scarring in severe cases[33]
  3. Secondary infections compound damage: Bacterial colonization can occur in open lesions[34]

Recovery Timeline and Expectations

Hair regrowth follows predictable patterns once inflammation resolves. Studies tracking 100 patients found:

  • Week 2-4: Inflammation subsides, itching decreases
  • Week 4-8: New vellus (fine) hairs appear
  • Week 8-12: Terminal hair growth resumes
  • Month 3-6: Full density recovery in many cases[9]

Permanent loss occurs only with prolonged untreated inflammation lasting over 6 months or repeated severe infections[35].

📚 Related Resource

For comprehensive application strategies, explore: Applying Eczema Cream to the Scalp

Treating Cradle Cap in Babies

Infantile seborrheic dermatitis, commonly called cradle cap, is common in infants during their first year of life[28]. While visually concerning to parents, it rarely causes discomfort to infants and typically resolves spontaneously by 12 months.

Understanding Cradle Cap Development

Research suggests multiple contributing factors:

  • Maternal hormones stimulate sebaceous gland activity[36]
  • Immature skin barrier function allows yeast proliferation
  • Genetic predisposition may play a role[36]

Safe Treatment Approaches

A Cochrane review of infantile seborrheic dermatitis treatments found limited high-quality evidence, but clinical experience supports graduated approach[28]:

Mild cases (less than 30% scalp involvement):

  1. Apply mineral oil or petroleum jelly to soften scales
  2. Leave for 15-60 minutes
  3. Gently brush with soft toothbrush
  4. Wash with mild baby shampoo

Moderate to severe cases:

  • Ketoconazole 2% shampoo twice weekly (minimal systemic absorption documented)[37]
  • Hydrocortisone 1% cream for inflamed areas, maximum 7 days
  • Avoid olive oil, which can worsen symptoms by promoting yeast growth[38]

Important Note: If cradle cap persists beyond 12 months or spreads to body folds, consider evaluation for atopic dermatitis, which affects up to 25% of children[39].

📚 Related Resource

Learn more about protecting your child's skin: Baby Eczema: Protect Your Child With These 7 Tips

Long-term Prevention Strategies

Preventing scalp eczema flares requires addressing both internal and external factors. Comprehensive prevention strategies, including proper skin care, trigger avoidance, and adjunctive therapies, can help reduce flare frequency[40].

Hair Care Modifications

Research-backed hair care adjustments include:

  • Washing frequency: Regular but not excessive washing helps maintain scalp health[40]
  • Water temperature: Cool to lukewarm water prevents barrier disruption
  • Drying technique: Pat dry and use cool setting on hairdryers to prevent irritation
  • Product selection: SLS-free shampoos may reduce irritation in sensitive individuals[40]

Dietary Considerations

While evidence remains mixed, certain dietary modifications show promise:

  • Omega-3 fatty acids: Evidence for benefit in eczema remains limited[40]
  • Probiotics: Evidence for routine use in established atopic dermatitis is insufficient[40]
  • Zinc supplementation: Evidence for benefit in atopic eczema is limited[40]

Stress Management Impact

Psychological stress can trigger flares in adults with scalp eczema[41]. Stress management interventions may provide benefit, though evidence varies[40].

When to See a Dermatologist

While many cases respond to over-the-counter treatments, certain situations require professional evaluation. Delaying appropriate care can lead to complications[40].

Red Flag Symptoms

Seek immediate medical attention for:

  • Signs of infection: Oozing, honey-colored crusts, or fever
  • Rapid spreading: Extension beyond scalp within days
  • Severe pain: Burning or stinging preventing sleep
  • Vision changes: If eczema affects eyelids or surrounding area

Treatment Failure Indicators

Consider dermatology referral when:

  • No improvement after 4 weeks of consistent OTC treatment
  • Symptoms worsen despite treatment
  • Hair loss exceeds 25% in affected areas
  • Quality of life significantly impacted

Dermatologists can offer advanced diagnostics including:

  • Dermoscopy: Magnified examination (trichoscopy) provides enhanced visualization of scalp structures and improves diagnostic accuracy for differentiating scalp conditions[27]
  • Patch testing: Can identify contact allergens in resistant cases[42]
  • Scalp biopsy: Definitive diagnosis when clinical picture unclear

Remember

Early professional intervention can help prevent complications and optimize treatment outcomes

What to Expect During Your Visit

Dermatologists typically follow systematic evaluation:

  1. Detailed history including trigger identification
  2. Full body skin examination (many patients have eczema elsewhere)[43]
  3. Microscopic evaluation of scale samples
  4. Discussion of prescription options based on severity
  5. Development of maintenance plan to prevent recurrence

Advanced Treatment Options

Dermatologists can prescribe treatments unavailable over-the-counter:

  • Compound formulations: Custom combinations targeting multiple mechanisms
  • Intralesional injections: For thick, resistant plaques
  • Excimer laser: Targeted UV therapy for localized patches
  • Biologics: For severe cases unresponsive to conventional therapy

Finding the right eczema cream designed for long-term use can bridge the gap between harsh prescription treatments and ineffective over-the-counter options. Modern formulations that address both inflammation and skin barrier health offer new hope for sustained remission.

Moving Forward with Confidence

Managing scalp eczema requires patience, consistency, and the right treatment approach for your specific type. The evidence clearly shows that combination therapies targeting both inflammation and underlying causes achieve the best outcomes. Whether you choose conventional treatments, natural remedies, or innovative dual-action eczema cream formulations, success comes from addressing root causes rather than just masking symptoms.

Remember that scalp eczema, while frustrating, is manageable. With proper treatment, many patients achieve significant improvement[44]. The key lies in identifying your type, understanding your triggers, and maintaining consistent treatment even after symptoms improve. Your scalp health journey may have been challenging so far, but armed with evidence-based strategies and appropriate medical support when needed, lasting relief is achievable.

References

  1. Polaskey MT, Chang CH, Daftary K, et al. The Global Prevalence of Seborrheic Dermatitis: A Systematic Review and Meta-Analysis. JAMA Dermatol. 2024;160(8):846-855. View Study
  2. Sanclemente G, Hernandez N, Chaparro D, et al. Epidemiologic features and burden of atopic dermatitis in adolescent and adult patients: A cross-sectional multicenter study. World Allergy Organ J. 2021;14(12):100611. View Study
  3. Cîrstea N, Radu A, Vesa C, et al. Current Insights on Treatment Adherence in Prevalent Dermatological Conditions and Strategies To Optimize Adherence Rates. Cureus. 2024;16(9):e69764. View Study
  4. Ali F, Vyas J, Finlay AY. Counting the Burden: Atopic Dermatitis and Health-related Quality of Life. Acta Derm Venereol. 2020;100(12):adv00161. View Study
  5. Kamata M, Tada Y. A Literature Review of Real-World Effectiveness and Safety of Dupilumab for Atopic Dermatitis. JID Innov. 2021;1(3):100042. View Study
  6. Lax SJ, Van Vogt E, Candy B, et al. Topical Anti-Inflammatory Treatments for Eczema: A Cochrane Systematic Review and Network Meta-Analysis. Clin Exp Allergy. 2024;54(12):960-972. View Study
  7. Park HK, Ha MH, Park SG, et al. Characterization of the fungal microbiota (mycobiome) in healthy and dandruff-afflicted human scalps. PLoS One. 2012;7(2):e32847. View Study
  8. Okokon EO, Verbeek JH, Ruotsalainen JH, et al. Topical antifungals for seborrhoeic dermatitis. Cochrane Database Syst Rev. 2015;(5):CD008138. View Study
  9. Blume-Peytavi U, Vogt A. Human hair follicle: reservoir function and selective targeting. Br J Dermatol. 2011;165(s2):13-17. View Study
  10. Hammer KA, Carson CF, Riley TV. In Vitro Activities of Ketoconazole, Econazole, Miconazole, and Melaleuca alternifolia (Tea Tree) Oil against Malassezia Species. Antimicrob Agents Chemother. 2000;44(2):467-9. View Study
  11. White TC, Findley K, Dawson TL, et al. Fungi on the skin: dermatophytes and Malassezia. Cold Spring Harb Perspect Med. 2015;4(8):a019802. View Study
  12. Palmer CN, Irvine AD, Terron-Kwiatkowski A, et al. Common loss-of-function variants of the epidermal barrier protein filaggrin are a major predisposing factor for atopic dermatitis. Nat Genet. 2006;38(4):441-6. View Study
  13. Jampuram H, Mahendra A, Gupta S. Clinical Evaluation, Patch Testing, and Quality of Life Assessment in Patients With Cosmetic Dermatitis. Cureus. 2024;16(12):e75503. View Study
  14. Lundov MD, Thyssen JP, Zachariae C, Johansen JD. Prevalence and cause of methylisothiazolinone contact allergy. Contact Dermatitis. 2010;63(3):164-167. View Study
  15. DaSilva SC, Sahu RP, Konger RL, et al. Increased skin barrier disruption by sodium lauryl sulfate in mice expressing a constitutively active STAT6 in T cells. Arch Dermatol Res. 2012;304(1):65-71. View Study
  16. Barnes L, Kaya G, Rollason V. Topical Corticosteroid-Induced Skin Atrophy: A Comprehensive Review. Drug Saf. 2015;38(5):493-509. View Study
  17. Kishibe M, Baida G, Bhalla P, et al. Important role of kallikrein 6 for the development of keratinocyte proliferative resistance to topical glucocorticoids. Oncotarget. 2016;7(43):69479-69488. View Study
  18. Eichenfield LF, Tom WL, Berger TG, et al. Guidelines of care for the management of atopic dermatitis: section 2. Management and treatment with topical therapies. J Am Acad Dermatol. 2014;71(1):116-32. View Study
  19. Sidbury R, Davis DM, Cohen DE, et al. Guidelines of care for the management of atopic dermatitis: section 3. Management and treatment with phototherapy and systemic agents. J Am Acad Dermatol. 2014;71(2):327-49. View Study
  20. Pink A. Best Systemic Treatments for Adults with Atopic Eczema over the Long Term (BEACON). ISRCTN. 2023. View Study
  21. Carson CF, Hammer KA, Riley TV. Melaleuca alternifolia (Tea Tree) oil: a review of antimicrobial and other medicinal properties. Clin Microbiol Rev. 2006;19(1):50-62. View Study
  22. Verallo-Rowell VM, Dillague KM, Syah-Tjundawan BS. Novel antibacterial and emollient effects of coconut and virgin olive oils in adult atopic dermatitis. Dermatitis. 2008;19(6):308-15. View Study
  23. Evangelista MT, Abad-Casintahan F, Lopez-Villafuerte L. The effect of topical virgin coconut oil on SCORAD index, transepidermal water loss, and skin capacitance in mild to moderate pediatric atopic dermatitis: a randomized, double-blind, clinical trial. Int J Dermatol. 2014;53(1):100-8. View Study
  24. Lambers H, Piessens S, Bloem A, et al. Natural skin surface pH is on average below 5, which is beneficial for its resident flora. Int J Cosmet Sci. 2006;28(5):359-70. View Study
  25. Goddard AL, Lio PA. Alternative, Complementary, and Forgotten Remedies for Atopic Dermatitis. Evid Based Complement Alternat Med. 2015;2015:676897. View Study
  26. Park JH, Park YJ, Kim SK, et al. Histopathological Differential Diagnosis of Psoriasis and Seborrheic Dermatitis of the Scalp. Ann Dermatol. 2016;28(4):427-434. View Study
  27. Rosina P, Zamperetti MR, Giovannini A, Girolomoni G. Videocapillaroscopy in the Differential Diagnosis between Psoriasis and Seborrheic Dermatitis of the Scalp. Dermatology. 2007;214(1):21-24. View Study
  28. Victoire A, Magin P, Coughlan J, van Driel ML. Interventions for infantile seborrhoeic dermatitis (including cradle cap). Cochrane Database Syst Rev. 2019;(3):CD011380. View Study
  29. Tollefson MM, Crowson CS, McEvoy MT, Kremers HM. Incidence of psoriasis in children: A population-based study. J Am Acad Dermatol. 2010;62(6):979-87. View Study
  30. Kragballe K, Hoffmann V, Ortonne JP, et al. Efficacy and safety of calcipotriol plus betamethasone dipropionate scalp formulation compared with calcipotriol scalp solution in the treatment of scalp psoriasis: a randomized controlled trial. Br J Dermatol. 2009;161(1):159-66. View Study
  31. Whiting DA. Chronic telogen effluvium: Increased scalp hair shedding in middle-aged women. J Am Acad Dermatol. 1996;35(6):899-906. View Study
  32. Ozlu E, Karadag AS. Telogen Effluvium. In: Hair and Scalp Disorders. IntechOpen; 2017. View Study
  33. Kligman AM. Pathologic Dynamics of Human Hair Loss. Arch Dermatol. 1961;83(2):175. View Study
  34. Rakowska A, Olszewska M, Rudnicka L. Telogen Effluvium. In: Atlas of Trichoscopy. London: Springer; 2012:237-244. View Study
  35. Trueb RM. Systematic approach to hair loss in women. J Dtsch Dermatol Ges. 2020;8(4):284-97. View Study
  36. Alexopoulos A, Kakourou T, Orfanou I, Xaidara A, Chrousos G. Retrospective Analysis of the Relationship Between Infantile Seborrheic Dermatitis and Atopic Dermatitis. Pediatr Dermatol. 2014;31(2):125-130. View Study
  37. Brodell RT, Patel S, Venglarcik JS, Moses D, Gemmel D. The safety of ketoconazole shampoo for infantile seborrheic dermatitis. Pediatr Dermatol. 1998;15(5):406-407. View Study
  38. Brunke S, Hube B. MfLIP1, a gene encoding an extracellular lipase of the lipid-dependent fungus Malassezia furfur. Microbiology. 2006;152(2):547-554. View Study
  39. Peroni DG, Piacentini GL, Bodini A, et al. Prevalence and risk factors for atopic dermatitis in preschool children. Br J Dermatol. 2008;158(3):539-43. View Study
  40. Sidbury R, Kodama S. Atopic dermatitis guidelines: Diagnosis, systemic therapy, and adjunctive care. Clin Dermatol. 2018;36(5):648-652. View Study
  41. Amano H, Negishi I, Akiyama H, Ishikawa O. Psychological Stress can Trigger Atopic Dermatitis in NC/Nga Mice: An Inhibitory Effect of Corticotropin-Releasing Factor. Neuropsychopharmacology. 2008;33(3):566-73. View Study
  42. Manuskiatti W, Maibach HI. 1- versus 2- and 3-day diagnostic patch testing. Contact Dermatitis. 1996;35(4):197-200. View Study
  43. Williams HC. Epidemiology of atopic dermatitis. Clin Exp Dermatol. 2000;25(7):522-9. View Study
  44. Spuls PI, Gerbens LAA, Simpson E, et al. Patient-Oriented Eczema Measure (POEM), a core instrument to measure symptoms in clinical trials: a Harmonising Outcome Measures for Eczema (HOME) statement. Br J Dermatol. 2017;176(4):979-84. View Study

About the Author: Jessica Arenas, Lead Research Analyst

Jessica makes sense of the numbers behind skin health. Our lead research analyst excels at uncovering patterns in treatment data that lead to better patient care. Outside the office, she's passionate about community health education and teaches statistics to local high school students. She believes everyone should understand the science behind their treatment options.