Seborrheic dermatitis affects nearly 1 in 20 people worldwide. That's according to a comprehensive meta-analysis of 121 studies involving over 1.2 million individuals[1]. Countless others may mistake their symptoms for simple dandruff or dry skin. The good news? Understanding the science behind this condition opens the door to effective, lasting relief.
Finding the right treatment often feels like solving a puzzle with missing pieces. Seborrheic dermatitis is chronic and relapsing. It requires ongoing management rather than a one-time cure[2]. Flaking returns days after washing. Redness appears despite careful skincare. Studies indicate that 30.9% of people with seborrheic dermatitis experience moderate-to-severe anxiety[3]. Your skin concerns are valid. Your journey matters.
This guide explores the science behind seborrheic dermatitis and provides evidence-based strategies for managing symptoms effectively. You'll learn why certain treatments work while others fail. You'll discover how to identify your personal triggers and prevent future flares. We'll examine treatments that address both inflammation and skin microbiome health. Every recommendation comes from peer-reviewed studies, not marketing claims.
Here's something interesting: prevalence varies dramatically by region. South Africa shows rates as high as 8.82%, while India has the lowest at 2.62%[1]. This variation suggests that personalized approaches often work better than one-size-fits-all solutions. Let's explore what the research reveals.
Key Takeaways
- Seborrheic dermatitis affects up to 5% of adults globally and peaks during infancy and middle age[6]
- Malassezia yeast plays a role but immune response and skin barrier function are equally important
- Antifungal treatments like ketoconazole show 73% improvement in clinical trials[11]
- Stress triggers flares in over 28% of patients[14] making stress management essential
- Long-term management requires combination approaches addressing multiple factors simultaneously
Table of Contents
What is Seborrheic Dermatitis?
Seborrheic dermatitis is a chronic inflammatory skin condition that causes scaly, flaky patches on areas rich in oil-producing glands. The scalp, face, and chest are most commonly affected. In infants, it mainly occurs on the scalp as yellowish, scaly patches with varying degrees of inflammation, known as "cradle cap"[4].
Unlike simple dry skin, seborrheic dermatitis involves an inflammatory response beneath the surface. The condition creates white or yellowish patches with greasy scales in areas rich in sebaceous glands[4]. These patches may itch, burn, or feel tender to the touch. Many people notice their symptoms worsen during winter months or periods of high stress.
The relationship between seborrheic dermatitis and dandruff causes confusion. Dandruff is regarded as a mild, non-inflammatory form of seborrheic dermatitis, causing white flakes without significant redness[5]. When inflammation accompanies the flaking, the condition moves beyond simple dandruff into true seborrheic dermatitis.
Clinical Insight: Seborrheic dermatitis affects up to 5% of the general population, with prevalence rising to 30-83% in HIV-infected individuals[6]. The condition shows a bimodal pattern, peaking in infants under 3 months and adults between ages 30-60.
Research from the Global Burden of Disease study reveals that seborrheic dermatitis cases increased by 53% between 1990 and 2021[7]. This rise likely reflects improved diagnosis rather than true increases in occurrence. Men develop the condition more frequently than women, and people with darker skin tones may experience different visual presentations[4].
What Causes Seborrheic Dermatitis?
Scientists have studied seborrheic dermatitis for over a century. Yet the exact cause remains surprisingly elusive. What we do know is that several factors work together to trigger flares. These include yeast overgrowth, immune system responses, skin barrier dysfunction, and genetic predisposition[8]. Understanding each piece helps explain why treatment works best when it targets multiple factors at once.
The Role of Malassezia Yeast
Malassezia yeast lives naturally on healthy skin. These lipid-dependent organisms thrive in areas with high sebum production. For decades, researchers believed Malassezia overgrowth directly caused seborrheic dermatitis. But here's where it gets interesting: the picture is far more complex than anyone initially thought.
Studies show that antifungal treatments reduce Malassezia counts and improve symptoms simultaneously[9]. This correlation supports the yeast connection. However, people without seborrheic dermatitis often carry similar Malassezia levels on their skin[10]. The yeast appears necessary but not sufficient to trigger the condition.
73%
Improvement rate with antifungal ketoconazole treatment in clinical trials[11]
Recent research suggests that the transition of Malassezia from yeast to hyphal (thread-like) form may enhance its ability to cause inflammation[12]. Patients with visible hyphae in skin samples showed significantly higher disease severity scores. Antifungal treatment eliminated both forms and improved symptoms.
Immune System and Skin Barrier Factors
If Malassezia were the whole story, everyone with the yeast would develop seborrheic dermatitis. They don't. So what makes the difference? Your immune system's response matters just as much as the yeast itself. People with seborrheic dermatitis show altered immune reactions, including changes in T-cell function and complement activation[8]. This explains why immunocompromised individuals often develop more severe cases.
Skin barrier dysfunction plays an equally important role. Research demonstrates increased transepidermal water loss in affected skin, meaning the protective barrier fails to hold moisture properly[13]. Understanding how moisturizers work helps explain why barrier repair is essential. This dysfunction allows yeast to penetrate deeper and trigger more inflammation.
Genetic factors influence susceptibility. Studies of patients with certain gene mutations show altered skin barrier function and increased seborrheic dermatitis risk[10]. While you cannot change your genes, understanding this connection helps explain why some family members share the condition.
📚 Related Resource
For deeper insights on how skin microbiome health affects eczema conditions, see our guide: The Skin Microbiome and Eczema Connection
Recognizing Seborrheic Dermatitis Symptoms
How seborrheic dermatitis looks depends on three things: your age, your skin tone, and where it appears on your body. Recognizing these patterns matters because the condition is often mistaken for psoriasis or atopic dermatitis. Getting the diagnosis right means getting the right treatment.
The scalp is ground zero for most people with seborrheic dermatitis. Symptoms can range from mild dandruff that barely bothers you to thick, stubborn crusts covering large areas of your scalp[4]. Here's what to watch for:
- White or yellowish flakes on hair and shoulders
- Greasy patches with redness underneath
- Itching that worsens with stress or weather changes
- Temporary hair thinning in severely affected areas
Facial seborrheic dermatitis typically affects specific zones. The eyebrows, sides of the nose, forehead, and areas around the ears show involvement most often[14]. Eyelid margins may become red and crusty, a presentation called seborrheic blepharitis.
| Location | Common Symptoms | Appearance |
|---|---|---|
| Scalp | Flaking, itching, greasy scales | White to yellow flakes, pink patches |
| Face | Redness, scaling around nose and eyebrows | Salmon-colored patches with fine scales |
| Chest | Red patches, greasy scales | Ring-shaped or petal-like patterns |
| Body Folds | Redness, less scaling, moist appearance | Glazed, inflamed skin |
Skin tone affects how seborrheic dermatitis appears visually. People with lighter skin typically see red, raised patches. Those with darker skin may notice lighter or darker discoloration compared to surrounding skin, sometimes without obvious redness[5]. The scaling pattern in darker skin sometimes appears flower-like or petal-shaped.
Symptoms follow a relapsing-remitting pattern. Flares may last weeks or months, then improve spontaneously. Many people notice seasonal patterns, with symptoms worsening in winter and early spring[4]. Warm, humid weather often brings improvement.
Common Triggers That Cause Flare-Ups
What sets off your flares? This is one of the most important questions you can answer. Understanding your personal triggers gives you real power over this condition. And research has identified several factors that commonly make symptoms worse.
Here's encouraging news: a study of 166 patients found that 83.1% could identify specific triggers for their flares[14]. That means most people can learn what to avoid. The most frequently reported aggravating factors included:
- Seasonal/weather changes: 34.9% of patients
- Emotional stress or sleep deprivation: 28.3%
- Cosmetic products: 21.7%
- Sweat and humidity: 14.5%
- Sun exposure: 14.5%
- Certain foods: 12.0%
Stress deserves special attention as a trigger. Clinical research confirms that seborrheic dermatitis is frequently preceded by stressful events[15]. The same study found that recognizing stress as a trigger predicted worse outcomes four months later. This bidirectional relationship means stress worsens symptoms, and visible symptoms increase stress.
Research Update: A cross-sectional study found that 30.9% of seborrheic dermatitis patients exhibited moderate-to-severe anxiety, highlighting the substantial psychological burden of this condition[3].
Dietary patterns may influence seborrheic dermatitis. A large study found that high fruit intake reduced risk by 25%, while a Western diet high in processed foods increased risk by 47% in females[16]. Patients commonly report that spicy foods, sweets, fried foods, and dairy products worsen their symptoms[17].
Certain medical conditions increase susceptibility. People with Parkinson's disease, HIV infection, and neurological conditions experience seborrheic dermatitis more frequently and severely[4]. Medications including lithium, certain immunosuppressants, and some psychiatric drugs can trigger or worsen flares[8].
How Doctors Diagnose Seborrheic Dermatitis
Most cases of seborrheic dermatitis don't require lab tests or biopsies. Dermatologists typically recognize the characteristic pattern of scaling in sebum-rich areas just by looking. That's good news for you: diagnosis is usually straightforward.
During your visit, expect questions about symptom duration, triggers you've noticed, and treatments you've already tried. Your doctor will examine where the scaling appears, what the scales look like, and whether there are any signs of secondary infection[4]. This examination usually takes just a few minutes.
Several conditions resemble seborrheic dermatitis and require differentiation. Understanding the different types of dermatitis helps guide proper treatment:
| Condition | Key Differences |
|---|---|
| Psoriasis | Thicker, silvery scales; often affects elbows, knees; nail changes common |
| Atopic Dermatitis | More itchy; affects arm and leg creases; often childhood onset |
| Contact Dermatitis | Tied to specific exposure; irregular borders; may have vesicles |
| Tinea (Fungal Infection) | Ring-shaped; advancing border; responds to different antifungals |
| Rosacea | Central face redness; flushing; no significant scaling |
Some patients have "sebopsoriasis," where features of both seborrheic dermatitis and psoriasis overlap. This hybrid presentation affects the scalp most commonly and may require treatments targeting both conditions[18].
In severe or atypical cases, doctors may perform a skin biopsy. Histological examination reveals characteristic findings including spongiotic dermatitis with inflammatory cell infiltrates[4]. HIV testing should be considered in cases of sudden, severe onset, as seborrheic dermatitis can be an early indicator of immunodeficiency[4].
Proven Treatment Options
Effective treatment addresses multiple aspects of seborrheic dermatitis at once. Most approaches do one of three things: target Malassezia yeast, reduce inflammation, or remove accumulated scales. Here's the key insight: combining strategies typically works better than any single approach alone.
Antifungal Treatments
Antifungal agents remain the cornerstone of seborrheic dermatitis treatment. They work by reducing Malassezia populations, which in turn decreases the inflammation triggered by the yeast. You have multiple options depending on whether you're treating your scalp, face, or both.
Ketoconazole shows the strongest evidence among antifungal options. A randomized trial of 331 patients found that 2% ketoconazole shampoo achieved 73% improvement in dandruff severity, significantly outperforming 1% zinc pyrithione (67% improvement)[11]. Recurrence rates were also lower with ketoconazole (23% vs 39%).
2-4 weeks
Typical time to see significant improvement with antifungal treatment[19]
Other effective antifungal options include:
- Ciclopirox: Comparable effectiveness to ketoconazole; reduces relapse for up to 12 weeks[19]
- Zinc pyrithione: Available over-the-counter; effective for mild cases[20]
- Selenium sulfide: Reduces dandruff; may cause scalp discoloration rarely[21]
For scalp treatment, ciclopirox shampoo is recommended to be used two to three times per week for clearance, then weekly or every other week for prophylaxis[21]. Daily use during flares typically transitions to twice-weekly maintenance once symptoms improve. Some patients also benefit from targeted scalp application of anti-inflammatory creams between washes.
Anti-Inflammatory Options
Anti-inflammatory treatments provide rapid relief from redness, itching, and swelling. Options range from mild over-the-counter preparations to prescription-strength medications.
Topical corticosteroids remain effective for short-term flare management. A Cochrane review of 36 trials found that steroids improved symptoms compared to placebo, with no significant difference between mild and strong formulations in the short term[22]. However, long-term facial use risks skin thinning, telangiectasias, and rebound flares.
Calcineurin inhibitors (tacrolimus, pimecrolimus) offer safer alternatives for long-term facial treatment. Studies show improvement within two weeks, with lower relapse rates than corticosteroids[23]. These medications do not cause skin atrophy, making them suitable for maintenance therapy[24].
Newer treatment options continue emerging. The FDA recently approved roflumilast foam, a phosphodiesterase-4 inhibitor, for seborrheic dermatitis[25]. This non-steroidal option shows promise for patients seeking alternatives to traditional therapies.
Combination Approaches: Addressing Multiple Factors Simultaneously
Seborrheic dermatitis involves multiple pathological processes: Malassezia overgrowth, inflammation, and skin barrier dysfunction. Single-target treatments often fall short because they address only one piece of the puzzle. This explains why many patients cycle through antifungals and steroids without achieving lasting control.
Combination formulations that target multiple mechanisms simultaneously show particular promise for chronic management. The ideal approach addresses:
- Malassezia control to reduce yeast-triggered inflammation
- Anti-inflammatory action to calm redness and itching
- Barrier repair to restore skin's protective function
- Microbiome support to maintain healthy skin flora balance
One formulation that addresses all four factors is SmartLotion, which combines low-dose hydrocortisone (0.75%) with sulfur (0.5%) and other barrier-supporting ingredients like glycerin and petrolatum. This specific combination is particularly relevant for seborrheic dermatitis for several reasons.
Sulfur targets Malassezia directly. Research shows sulfur has documented antimicrobial activity against Malassezia species, the yeast implicated in seborrheic dermatitis. Unlike standalone antifungal shampoos that wash off quickly, a leave-on formulation maintains continuous contact with affected skin.
The sulfur-hydrocortisone combination prevents steroid complications. A landmark 15-year study followed 300 patients using this exact combination on facial skin. The results showed zero incidence of steroid acne, rebound phenomenon, or skin atrophy despite continuous application. This finding is remarkable because facial skin is the most vulnerable to steroid side effects, yet the sulfur appears to protect against these complications through multiple pathways.
15 years
Duration of facial use with zero steroid complications in 300-patient study
Long-term safety matters for chronic conditions. Seborrheic dermatitis is relapsing-remitting by nature. Many patients need ongoing treatment for months or years. Traditional corticosteroids become problematic with extended facial use, forcing patients to cycle on and off treatment. A formulation designed for continuous use without accumulating side effects changes this equation.
Barrier repair supports lasting improvement. The glycerin and petrolatum components address the transepidermal water loss documented in seborrheic dermatitis. Research shows petrolatum upregulates antimicrobial peptides and barrier proteins, while glycerin activates aquaporin-mediated hydration. These effects complement the anti-inflammatory and antimicrobial actions.
Dr. Harlan, who developed this formulation, has created a specific seborrheic dermatitis treatment protocol based on clinical experience with thousands of patients. The protocol addresses both acute flares and maintenance therapy, recognizing that different phases of the condition require different approaches.
When to Consider Combination Therapy: Patients who have tried antifungal shampoos and topical steroids separately without lasting success may benefit from a combination approach. Those with facial involvement, where steroid safety is a concern, are particularly good candidates for formulations designed for long-term use.
📚 Related Resource
Learn more about choosing treatments for facial dermatitis: Facial Eczema Care Guide
Natural Remedies and Home Care
Natural approaches complement medical treatment and may provide relief for mild cases. Research supports several botanical and lifestyle interventions, though evidence varies in quality.
Aloe vera demonstrates the strongest evidence among natural remedies. A double-blind, placebo-controlled study of 44 adults found that 30% aloe vera ointment produced 58% improvement compared to 15% with placebo[26]. Both patient and physician assessments confirmed reduced itching, scaliness, and affected area size.
Tea tree oil possesses antifungal properties against Malassezia. A placebo-controlled study found that 5% tea tree oil shampoo significantly reduced dandruff symptoms[21]. Always dilute tea tree oil before skin application, as concentrated forms may cause irritation.
Other natural options showing potential include:
- Honey: Raw honey has antimicrobial properties and may reduce scaling when applied as a mask
- Coconut oil: Mixed results; moisturizing but may feed Malassezia in some people
- Apple cider vinegar: Anecdotal reports of reduced flaking; dilute before applying to avoid irritation
- Omega-3 fatty acids: May help reduce inflammation when taken as supplements
A word of caution: natural doesn't always mean safe for everyone. Some people find that gentle moisturizers help soothe affected areas between treatments. Others discover that oil-based products actually worsen symptoms by providing nutrients for Malassezia yeast. Pay attention to how your skin responds.
Gentle skincare practices support healing regardless of treatment choice. Avoid harsh soaps and detergents that strip natural oils. Choose fragrance-free products designed for sensitive skin. An effective eczema cream formulated for sensitive skin can help maintain barrier function between flares.
Dietary modifications may reduce flares for some people. The gut-skin axis connects intestinal health with skin inflammation. A clinical trial found that shampoo containing heat-killed Lacticaseibacillus paracasei improved scalp health by reducing dandruff and sebum secretion[28].
Long-Term Management Strategies
Here's the reality that many people resist hearing: seborrheic dermatitis requires ongoing management rather than one-time treatment. Once you accept this, everything changes. You stop searching for a magic cure. You start building sustainable routines that actually work.
Weeks 1-2: Active Treatment Phase
Use medicated products daily or as directed. Expect initial improvement in flaking and redness[19].
Weeks 3-4: Stabilization
Symptoms continue improving. Begin transitioning to maintenance frequency.
Ongoing: Maintenance Phase
Use medicated shampoos 1-2 times weekly. Monitor for early signs of flares[21].
As Needed: Flare Management
Return to active treatment frequency when symptoms recur. Early intervention prevents severe flares.
Maintenance therapy significantly extends remission periods. Studies comparing zinc pyrithione and selenium sulfide shampoos for maintenance found no significant difference in relapse timing between products[27]. Consistency matters more than specific product choice.
Stress management deserves attention in long-term planning. Research confirms that stress both triggers and worsens seborrheic dermatitis[15]. Consider incorporating relaxation techniques like meditation, deep breathing, or regular exercise into your routine.
Keeping a symptom diary helps identify personal patterns. Track flares alongside potential triggers including stress levels, sleep quality, dietary changes, and weather conditions. Over time, patterns emerge that guide individualized prevention strategies.
When to See a Dermatologist
Can you handle seborrheic dermatitis on your own? Often, yes. Many people manage mild cases successfully with over-the-counter products and good skincare habits. But there are clear signals that it's time to call in a professional.
Consult a dermatologist if you experience:
- Symptoms that persist despite 2-4 weeks of over-the-counter treatment
- Severe scaling, crusting, or widespread involvement
- Signs of infection (oozing, increased pain, spreading redness)
- Significant impact on quality of life or daily activities
- Uncertainty about diagnosis
Prescription options provide stronger results for resistant cases. Dermatologists can offer higher-strength antifungals, prescription-only anti-inflammatory treatments, and combination therapies[25].
The psychological impact of seborrheic dermatitis justifies seeking help even for mild cases. Studies show that visible skin conditions significantly affect self-esteem and social interactions[3]. Dermatologists understand these concerns and can provide both medical treatment and emotional support.
Important: Sudden, severe seborrheic dermatitis in adults may indicate underlying conditions including HIV infection or neurological disorders[4]. Prompt evaluation ensures appropriate testing and management.
Living Well with Seborrheic Dermatitis
Seborrheic dermatitis isn't going to disappear forever. But with the right approach, you can minimize its impact on your daily life. Most people find that building a sustainable routine makes all the difference. The key word here is sustainable: something you can actually stick with.
Keep your morning and evening skincare routines simple. Complicated regimens tend to fall apart within weeks. Choose gentle, fragrance-free cleansers and moisturizers formulated for sensitive skin. A well-formulated eczema cream can help maintain your skin barrier function and reduce how often flares occur.
Practical tips for daily management include:
- Wash affected areas gently without scrubbing
- Apply moisturizer immediately after washing while skin is damp
- Avoid touching or picking at affected areas
- Choose natural-fiber clothing that breathes
- Protect skin from extreme temperatures
Social support matters. Connecting with others who understand seborrheic dermatitis reduces isolation and provides practical tips. Online communities and support groups offer opportunities to share experiences and coping strategies.
One more thing: flares do not mean you've failed. Even people with excellent management routines experience periodic worsening. It's the nature of the condition. Having a plan for these moments reduces anxiety and lets you respond quickly. Keep your maintenance products accessible. Know when to increase treatment intensity. And don't beat yourself up when symptoms return.
Seborrheic dermatitis affects millions of people worldwide. You're not alone in this. And more importantly, effective management is absolutely achievable. The key lies in understanding how Malassezia yeast, immune responses, and skin barrier function all interact. When you address multiple factors together, results improve dramatically.
Your path to clearer skin starts with what you've just learned. Whether you choose medicated shampoos, natural remedies, prescription therapies, or a combination approach, one thing matters above all else: consistency. Track your triggers. Maintain good skincare habits. Seek professional help when home treatments aren't enough.
Research keeps advancing. New treatment options emerge every year. Better understanding of this condition means better outcomes for patients. With proper care, seborrheic dermatitis transforms from overwhelming to manageable. Your skin is worth the effort. So is your peace of mind.
References
- Polaskey MT, Chang CH, Daftary K, et al. "The Global Prevalence of Seborrheic Dermatitis: A Systematic Review and Meta-Analysis." JAMA Dermatology, vol. 160, no. 8, 2024, pp. 846-855. View Study
- Borda LJ, Wikramanayake TC. "Seborrheic Dermatitis and Dandruff: A Comprehensive Review." Journal of Clinical and Investigative Dermatology, vol. 3, no. 2, 2015. View Study
- Taş Aygar G, Karataş H, Kaya E, Borazan NŞ, Kartal SP. The psychosocial dimensions of seborrheic dermatitis: A cross-sectional study on anxiety, personality, and quality of life. PLoS One. 2025;20(8):e0331030. View Study
- Dall'Oglio F, Nasca MR, Gerbino C, Micali G. An Overview of the Diagnosis and Management of Seborrheic Dermatitis. Clin Cosmet Investig Dermatol. 2022;15:1537-1548. View Study
- Thomas LM, Khasraghi AH. Topical Treatment of Seborrhoeic Dermatitis and Dandruff: An Overview. Ann Trop Med Public Health. 2020;23:S231823. View Study
- Barabanov AL, Barabanova EM. Seborrheic Dermatitis in Immunocompetent and HIV-Infected Persons. Dermatovenerology Cosmetology. 2023;9(3). View Study
- Hao Z, Zhao X, Chen J, Li B, Xu W, Zhang L. Global and Regional Burden of Seborrheic Dermatitis: Trends in Incidence and DALYs, 1990-2021. Clin Cosmet Investig Dermatol. 2025;18:1389-1400. View Study
- Wikramanayake TC, Borda LJ, Miteva M, Paus R. Seborrheic dermatitis—Looking beyond Malassezia. Exp Dermatol. 2019;28(9):991-1001. View Study
- DeAngelis YM, Gemmer CM, Kaczvinsky JR, Kenneally DC, Schwartz JR, Dawson TL. Three etiologic facets of dandruff and seborrheic dermatitis: Malassezia fungi, sebaceous lipids, and individual sensitivity. J Invest Dermatol Symp Proc. 2005;10(3):295-297. View Study
- Chang CH, Chovatiya R. More yeast, more problems?: reevaluating the role of Malassezia in seborrheic dermatitis. Arch Dermatol Res. 2024;316(4):100. View Study
- Piérard-Franchimont C, Goffin V, Decroix J, Piérard GE. A multicenter randomized trial of ketoconazole 2% and zinc pyrithione 1% shampoos in severe dandruff and seborrheic dermatitis. Skin Pharmacol Appl Skin Physiol. 2002;15(6):434-441. View Study
- Li J, Feng Y, Liu C, et al. Presence of Malassezia Hyphae Is Correlated with Pathogenesis of Seborrheic Dermatitis. Microbiol Spectr. 2022;10(1):e0116921. View Study
- Turner GA, Hoptroff M, Harding CR. Stratum corneum dysfunction in dandruff. Int J Cosmet Sci. 2012;34(4):298-306. View Study
- Araya M, Kulthanan K, Jiamton S. Clinical Characteristics and Quality of Life of Seborrheic Dermatitis Patients in a Tropical Country. Indian J Dermatol. 2015;60(5):519. View Study
- Misery L, Touboul S, Vinçot C, et al. Stress and seborrheic dermatitis. Ann Dermatol Venereol. 2007;134(11):833-837. View Study
- Sanders MGH, Pardo LM, Ginger RS, Kiefte-de Jong JC, Nijsten T. Association between Diet and Seborrheic Dermatitis: A Cross-Sectional Study. J Invest Dermatol. 2019;139(1):108-114. View Study
- Alshaebi MF, Zahed L, Osaylan M, Sulaimani S, Albahlool A, Abduljabbar MH, Hariri J. Association Between Diet and Seborrheic Dermatitis: A Case-Control Study. Cureus. 2023;15(11):e48782. View Study
- Ayala F. Clinical presentation of psoriasis. Reumatismo. 2011;59 Suppl 1:40-45. View Study
- Naldi L, Diphoorn J. Seborrhoeic dermatitis of the scalp. BMJ Clin Evid. 2015;2015:1713. View Study
- Mangion SE, Holmes AM, Roberts MS. Targeted Delivery of Zinc Pyrithione to Skin Epithelia. Int J Mol Sci. 2021;22(18):9730. View Study
- Berk T, Scheinfeld N. Seborrheic Dermatitis. P T. 2010;35(6):348-352. View Study
- Oksanen T, Kastarinen H, Kiviniemi V, Airola K, Peura P, Okokon EO, Verbeek JH. Topical anti-inflammatory agents for seborrhoeic dermatitis of the face or scalp. Cochrane Database Syst Rev. 2014;(5):CD009446. View Study
- Kim BS, Kim SH, Kim MB, Oh CK, Jang HS, Kwon KS. Treatment of Facial Seborrheic Dermatitis with Pimecrolimus Cream 1%: An Open-Label Clinical Study in Korean Patients. J Korean Med Sci. 2007;22(5):868-872. View Study
- Kim GK, Del Rosso JQ. Topical Pimecrolimus 1% Cream in the Treatment of Seborrheic Dermatitis. J Clin Aesthet Dermatol. 2013;6(2):29-35. View Study
- Vidal SI, Menta N, Green L. Child and Adult Seborrheic Dermatitis: A Narrative Review of the Current Treatment Landscape. Dermatol Ther (Heidelb). 2025;15(3):599-613. View Study
- Vardy DA, Cohen AD, Tchetov T, Medvedovsky E, Biton A. A double-blind, placebo-controlled trial of an Aloe vera (A. barbadensis) emulsion in the treatment of seborrheic dermatitis. J Dermatolog Treat. 1999;10(1):7-11. View Study
- Hasanbeyzade S. The Effects of Zinc Pyrithione and Selenium Disulfide Shampoos on the Lesion-free Period After Treatment in Patients with Seborrheic Dermatitis. J Clin Aesthet Dermatol. 2023;16(5):40-42. View Study
- Tsai WH, Fang YT, Huang TY, Chiang YJ, Lin CG, Chang WW. Heat-killed Lacticaseibacillus paracasei GMNL-653 ameliorates human scalp health by regulating scalp microbiome. BMC Microbiol. 2023;23:121. View Study